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Table S1. Demographic and disease characteristics at baseline randomized data set. Table S3. Table S4. Mean changes in other efficacy endpoints from baseline to week 24 randomized data set. Adverse events were similar between groups and consistent with previous studies. Key inclusion criteria were: HbA1c, 7.

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The majority of women with polycystic ovary syndrome PCOS have insulin resistance or high insulin levels. Too much insulin generates inflammation, which in turn can lead to the development of type 2 diabetes, obesity, dyslipidemia, and non-alcoholic fatty-liver disease. Insulin resistance is also one of the root causes of PCOS. It works by improving insulin sensitivity which in turn decreases insulin resistance and glucose production in your body. Women with PCOS often have low ovulation rates and irregular menstrual cycles, making it difficult to conceive. It can also treat diabetes that develops during pregnancy gestational diabetes and delay or prevent full-blown diabetes from developing in women who are overweight.

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  • That leaflet answers some common questions about this product.
  • Regular assessment of renal function is necessary see section 4.
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How does it work mechanism of action? Insulin is a hormone produced by the pancreas that controls glucose levels in blood by reducing the amount of glucose made by the liver and by increasing the removal of glucose from the blood by muscle and fat tissues. As a result, insulin causes blood glucose levels fall.

   
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Elisabeth (taken for 2 to 4 years) 25.07.2017

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Rapid decreases in fasting plasma glucose levels were observed by week 1, which continued until week 8, and were maintained for the duration of the study. In the U.

   
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Josef (taken for 1 to 6 years) 21.02.2018

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