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High-dose progestogen-only contraceptives, such as DMPA, inhibit follicular development and prevent ovulation as their primary mechanism of action. Decreased levels of FSH inhibit follicular development, preventing an increase in estradiol levels. Inhibition of follicular development and the absence of a LH surge prevent ovulation. However, because DMPA is highly effective in inhibiting ovulation and sperm penetration, the possibility of fertilization is negligible. MPA suppresses the hypothalamic-pituitary-adrenal HPA and hypothalamic-pituitary-gonadal HPG axes at sufficient dosages, resulting decreased levels of gonadotropins, androgens, estrogens, adrenocorticotropic hormone ACTH, and cortisol, as well as levels of sex hormone-binding globulin SHBG.

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